STROKE-PRESENTATION, LACUNAR INFARCT

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SUMMARY

Occlusive arteriolar or small artery disease leading to small lesions in the putamen, pons, thalamus, caudate, internal capsule:

1. 23% with preceding TIA & strong correlation with HTN[1]

PRESENTATION
2. Pure hemiplegia (with no sensory dysfunction): lesion in the posterior limb of the internal capsule/lower pons[2]

3. Pure hemisensory stroke (with no motor dysfunction): lesion in the sensory nucleus of the thalamus[3]

4. Sensory motor stroke

5. Ataxic hemiparesis (ataxia ipsilateral to hemiparesis): lesion in the upper pons[4]

6. Clumsy hand-dysarthria syndrome: lesion in the upper pons[5]

7. Multiple bilateral frontal lobe lacunae can result in pseudobulbar palsy-emotional lability with uninhibited crying or laughter and UMN signs (brisk jaw jerk, hyper-reflexia, and Babinski sign)


Reference(s)

  1. Fisher CM. Lacunar stroke and infarcts- a review. Neurology 1982; 32: 871-876.
  2. Fisher CM. Lacunar stroke and infarcts- a review. Neurology 1982; 32: 871-876.
  3. Fisher CM. Lacunar stroke and infarcts- a review. Neurology 1982; 32: 871-876.
  4. Fisher CM. Lacunar stroke and infarcts- a review. Neurology 1982; 32: 871-876.
  5. Fisher CM. Lacunar stroke and infarcts- a review. Neurology 1982; 32: 871-876.


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Hannaman, R. A., Bullock, L., Hatchell, C. A., & Yoffe, M. (2016). Internal medicine review core curriculum, 2017-2018. CO Springs, CO: MedStudy.
Therapeutic Guidelines. Melbourne: Therapeutic Guidelines Limited. https://www.tg.org.au [Accessed 2021].