Difference between revisions of "AUTOREGULATION-CAUSES OF BRADYCARDIA"

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==Reference(s)==
==Reference(s)==
Barrett, K.E., Barman, S.M., Boitano, S., Brooks, H.L., Weitz, M., Brian Patrick Kearns, Ganong, W.F. and Mcgraw-Hill Education (Firm (2016). Ganong’s review of medical physiology. 25th ed. New York: Mcgraw Hill Education.
Barrett, K.E., Barman, S.M., Brooks, H.L., X, J. and Ganong, W.F. (2019). Ganong’s review of medical physiology. 26th ed. New York: Mcgraw-Hill Education  
<br/>Hall, J.E. and Hall, M.E. (2020). Guyton And Hall Textbook Of Medical Physiology. 14th ed. S.L.: Elsevier - Health Science.
<br/>West, J.B. and Luks, A.M. (2021). West’s Pulmonary Pathophysiology. Lippincott Williams & Wilkins.


[[Category:Autoregulation]]
[[Category:Autoregulation]]
[[Category:Physiology]]
[[Category:Physiology]]

Latest revision as of 02:30, 21 March 2023

SUMMARY

Tip: CO = HR x SV

1. Increased CO: due to increased arterial baroreceptor activity.

2. Increased venous return: due to increased atrial baroreceptor activity.

3. Expiration: due to increased venous return & increased atrial baroreceptor activity.

4. Emotions (fear): due to limbic cortex inhibiting the vasomotor area.

5. Hypothermia.

6. Raised ICP: due to Cushing's response.


Reference(s)

Barrett, K.E., Barman, S.M., Brooks, H.L., X, J. and Ganong, W.F. (2019). Ganong’s review of medical physiology. 26th ed. New York: Mcgraw-Hill Education