Difference between revisions of "RENIN ANGIOTENSIN SYSTEM-TISSUE SYSTEM"

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==Reference(s)==
==Reference(s)==
Barrett, K.E., Barman, S.M., Boitano, S., Brooks, H.L., Weitz, M., Brian Patrick Kearns, Ganong, W.F. and Mcgraw-Hill Education (Firm (2016). Ganong’s review of medical physiology. 25th ed. New York: Mcgraw Hill Education.
Barrett, K.E., Barman, S.M., Brooks, H.L., X, J. and Ganong, W.F. (2019). Ganong’s review of medical physiology. 26th ed. New York: Mcgraw-Hill Education  
<br/>Hall, J.E. and Hall, M.E. (2020). Guyton And Hall Textbook Of Medical Physiology. 14th ed. S.L.: Elsevier - Health Science.
<br/>West, J.B. and Luks, A.M. (2021). West’s Pulmonary Pathophysiology. Lippincott Williams & Wilkins.


[[Category:Renin Angiotensin System]]
[[Category:Renin Angiotensin System]]
[[Category:Physiology]]
[[Category:Physiology]]

Latest revision as of 02:30, 21 March 2023

SUMMARY

1. Independent of circulating RAS.

2. Intrarenal RAS: most important tissue RAS.

3. Angiotensin II in renal interstitial fluid is approximately 1000x higher than in the systemic system.

4. Very sensitive to sodium intake.

5. Causes vasoconstriction and anti-natriuresis.


Reference(s)

Barrett, K.E., Barman, S.M., Brooks, H.L., X, J. and Ganong, W.F. (2019). Ganong’s review of medical physiology. 26th ed. New York: Mcgraw-Hill Education