ACUTE KIDNEY INJURY (AKI)-POST RENAL AKI CAUSES

From NeuroRehab.wiki

Revision as of 20:03, 13 March 2023 by Dr Appukutty Manickam (talk | contribs) (Imported from text file)
(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)

SUMMARY

1. In males: prostatic hypertrophy and stones are the usual causes. History (males/females): urinary urgency, frequency, poor stream, post void dribbling, incontinence. Supra-pubic pain (severe if acute). Examination: distended bladder.

2. Intratubular obstruction can be caused by uric acid precipitation, oxalate depositions, hypercalcemia with intrarenal deposits (remember Type 1 distal RTA!).

3. Multiple myeloma with light chains, myeloma cast nephropathy.

4. Drugs: methotrexate, indinavir, acyclovir, ganciclo­vir, and sulfa antibiotics (which can crystallize in the urine).


Reference(s)

Wilkinson, I., Furmedge, D. and Sinharay, R. (2017). Oxford handbook of clinical medicine. Oxford: Oxford University Press. Get it on Amazon.
Feather, A., Randall, D. and Waterhouse, M. (2020). Kumar And Clark’s Clinical Medicine. 10th ed. S.L.: Elsevier Health Sciences. Get it on Amazon.
Hannaman, R. A., Bullock, L., Hatchell, C. A., & Yoffe, M. (2016). Internal medicine review core curriculum, 2017-2018. CO Springs, CO: MedStudy.
Therapeutic Guidelines. Melbourne: Therapeutic Guidelines Limited. https://www.tg.org.au [Accessed 2021].

Retrieved from "https://www.neurorehab.wiki/index.php?title=ACUTE_KIDNEY_INJURY_(AKI)-POST_RENAL_AKI_CAUSES&oldid=18010"