Difference between revisions of "CARDIAC OUTPUT-DETERMINANTS OF EDV"

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==Reference(s)==
==Reference(s)==
Barrett, K.E., Barman, S.M., Boitano, S., Brooks, H.L., Weitz, M., Brian Patrick Kearns, Ganong, W.F. and Mcgraw-Hill Education (Firm (2016). Ganong’s review of medical physiology. 25th ed. New York: Mcgraw Hill Education.
Barrett, K.E., Barman, S.M., Brooks, H.L., X, J. and Ganong, W.F. (2019). Ganong’s review of medical physiology. 26th ed. New York: Mcgraw-Hill Education  
<br/>Hall, J.E. and Hall, M.E. (2020). Guyton And Hall Textbook Of Medical Physiology. 14th ed. S.L.: Elsevier - Health Science.
<br/>West, J.B. and Luks, A.M. (2021). West’s Pulmonary Pathophysiology. Lippincott Williams & Wilkins.


[[Category:Cardiac Output]]
[[Category:Cardiac Output]]
[[Category:Physiology]]
[[Category:Physiology]]

Latest revision as of 02:30, 21 March 2023

SUMMARY

REDUCED END-DIASTOLIC VOLUME
1. Reduced ventricular compliance (e.g. MI, infiltrative disease).
2. Pericardial effusion.
3. Reduced venous return to heart, e.g. standing.

INCREASED END-DIASTOLIC VOLUME
4. Stronger atrial contraction.
5. Negative intra-thoracic pressure during inspiration.
6. Increased venous return to heart, e.g. muscle pumps during exercise.


Reference(s)

Barrett, K.E., Barman, S.M., Brooks, H.L., X, J. and Ganong, W.F. (2019). Ganong’s review of medical physiology. 26th ed. New York: Mcgraw-Hill Education