Difference between revisions of "MEDICATION-ATROPINE"

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1. Parasympathetic antagonist that increases HR by blocking vagal tone.<div>2. Indications: symptomatic bradycardia with haemodynamic compromise. A bradycardic patient with no pulse should be treated as for PEA.</div><div>3. SE: dysthymia, HTN, urinary retention, delirium, hyperthermia in large doses.</div><div><br></div><div>DOSAGE </div><div>4. Rapid boluses of 500-600 mcg every 3-5 min up to a maximum of 3 mg.</div><div>5. Do not give slowly or boluses &lt; 500 mcg as this may irritate vagal fibres and cause a paradoxical reduction in HR.</div><div>6. Contraindicated in heart transplant patients as denervated hearts do not respond and may cause a heart block. </div>
===== [[Summary Article|'''SUMMARY''']] =====
 
1. Parasympathetic antagonist that increases HR by blocking vagal tone.
<br/>2. Indications: symptomatic bradycardia with haemodynamic compromise. A bradycardic patient with no pulse should be treated as for PEA.
<br/>3. SE: dysthymia, HTN, urinary retention, delirium, hyperthermia in large doses.
<br/>
<br/>DOSAGE  
<br/>4. Rapid boluses of 500-600 mcg every 3-5 min up to a maximum of 3 mg.
<br/>5. Do not give slowly or boluses &lt; 500 mcg as this may irritate vagal fibres and cause a paradoxical reduction in HR.
<br/>
<br/>6. Contraindicated in heart transplant patients as denervated hearts do not respond and may cause a heart block.  


[[Category:Medication]]
[[Category:Medication]]
[[Category:Advanced Life Support]]
[[Category:Advanced Life Support]]

Latest revision as of 11:31, 1 January 2023

SUMMARY

1. Parasympathetic antagonist that increases HR by blocking vagal tone.
2. Indications: symptomatic bradycardia with haemodynamic compromise. A bradycardic patient with no pulse should be treated as for PEA.
3. SE: dysthymia, HTN, urinary retention, delirium, hyperthermia in large doses.

DOSAGE
4. Rapid boluses of 500-600 mcg every 3-5 min up to a maximum of 3 mg.
5. Do not give slowly or boluses < 500 mcg as this may irritate vagal fibres and cause a paradoxical reduction in HR.

6. Contraindicated in heart transplant patients as denervated hearts do not respond and may cause a heart block.